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development of IAA was thought to be probably on account of oxidase deamination of NMT (Barker et al., 1980), but was later on set up to be also partially by immediate deamination by MAO (Barker et al., 1982). Pretreatment Together with the MAO-I iproniazid in rat total Mind homogenates inhibited IAA development by eighty three%, NMT and DMT-NO for
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